If you have problems in sexual life, you will help the drug temporarily, but effective. But when the disease is expressed, then generic Viagra is powerless here. Feelings of pain can be slightly perceptible, and can be transported very hard and are usually located in the middle of the back. Also, pain can be accompanied by many muscle spasms. Also, pain due to pinched nerve can give to the front of the chest and abdominal cavity, bringing pain, both in standing and sitting position. Compression of the nerves in the thoracic region prevents transmission of nerve signals from the brain to the peripheral nervous system.
A person may experience numbness or tingling, which extend from the back to the fingers of the upper and lower extremities. If you have a pinched nerve in the thoracic spine, the body can develop neurological deficits. Compression of the nerves of the thoracic spine can lead to muscle weakness in the legs. In severe forms of the disease may develop a temporary paralysis of the lower nerve compression point.
What Is Nerve Damage
The role of the compression of the peripheral branches of the trigeminal nerve has become more understandable in light of the “portal” theory of pain. It was found that the pain in this disease is associated with selective loss of thick myelinated fibers responsible for the rapid holding of well-localized pain, and the inclusion of “trigger” of central control – antinociceptive systems of the brain stem, and the proliferation of fibers other than slow performing vaguely localized (protopathic) pain. Evidence of the formation of focus of pathological activity in the central nervous system is increased pain during stimulation of the auditory and optic nerves, negative emotions. The clinical manifestations of the disease occur in violation of the relation between pain and a hotbed of antinociceptive structures at the level of secondary and intermediate brain, which explains the high incidence of the disease in patients with cerebrovascular pathology, in which the relatively common and affects early oral parts of the brain stem.
Discussion about whether trigeminal neuralgia functional or structural disease, is already finished. An experimental and clinical studies have shown that after 3 – 6 months from the onset of the disease in the affected axons branch of the trigeminal nerve detect structural changes in the form of swelling, vacuolization and fragmentation which in the course of the disease progress and in later stages turn into granular disintegration. Central mechanisms of pain paroxysm implementation included a second under the influence pathologic afferentation from the periphery.
Thus, the idea of the formation of pockets of paroxysmal activity of neurons in the brain as a result of irritation of brain structures at low excitation threshold cortico-subcortical structures under the influence of endocrine and metabolic factors, circulatory disorders and immunological changes in trigeminal neuralgia is reasonable.
Causes of Nerve Damage
It occurs in women in 2 times more often than men. It occurs primarily between the ages of 40 to 50 years with men – at a younger age. It is a chronic disease that lasts for decades. Usually, the pain occurs on one side, and after some time (significant time period – from several months to several years) – and on the other. The most frequently affected second and third branch of the trigeminal nerve, using one or on both sides. The combined loss of these branches is usually observed on one of the affected parties.
In the pathogenesis of age are important, allergic and vascular factors. Aggravating factors include infection, hypothermia, poor dentures, trauma, etc. The paroxysms of pain usually appear alternately on different sides of the face. Only in individual patients have simultaneous development of paroxysm on both sides, but more often than not with predominance to any one of them? In 50% of cases in the acute phase of the disease are determined trigger points often in the nasolabial region, at least – in the lateral parts of the facial skin and in the mouth. Often they are arranged symmetrically on both sides, sometimes are migratory in nature. In most patients the pain at the exit point on the face of the affected branches of the trigeminal nerve hypoesthesia with areas of anesthesia, hyperesthesia with areas hyperpathia (usually in patients for the treatment of previously used destructive methods), pronounced vegetative disorders and asthenoneurotic reaction.
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